front 1 Explain the general features of the alternative, lectin, and classical complement pathways | back 1 no data |
front 2 How are the pathways initiated? | back 2 no data |
front 3 What intermediates are produced? | back 3 no data |
front 4 what is the end result of each (membrane attack complex)? | back 4 no data |
front 5 Predict consequences for individuals that lack specific complement components due to genetic anomalies (e.g. non-functional complement genes). | back 5 no data |
front 6 Describe the key properties of a substance that functions as an opsonin. | back 6 no data |
front 7 Explain the benefit of opsonization. | back 7 no data |
front 8 Identify the complement products that function as opsonins (C3b primarily). | back 8 no data |
front 9 Correctly identify complement cleavage products that function as anaphylatoxins (C3a and C5a primarily). | back 9 no data |
front 10 Describe the functions and beneficial properties of an anaphylatoxin. | back 10 no data |
front 11 Correctly list the 4 hallmark features of inflammation (heat, swelling, pain, redness) | back 11 no data |
front 12 describe some of the biological processes involved in causing inflammation. | back 12 no data |
front 13 Describe the source (liver) and the biological activities of mannose binding lectin and C-reactive protein. | back 13 no data |
front 14 Describe examples of inhibitors of the complement pathways and explain how they function. | back 14 no data |
front 15 Explain why it is important to have normal levels of inhibitory complement proteins. | back 15 no data |
front 16 Use your understanding and knowledge of these issues to speculate and theorize about the cause, treatment, and consequences of complement-related health issues. | back 16 no data |