front 1 ACTIVITY 1: NORMAL BREATHING
| back 1 BETWEEN 7.38 AND 7.42 |
front 2 ACTIVITY 1: NORMAL BREATHING
| back 2 YES, THE pH (7.38 - 7.42) WAS WITHIN THE NORMAL pH RANGE OF 7.35 AND 7.45 |
front 3 ACTIVITY 2: HYPERVENTILATION
| back 3 40 |
front 4 ACTIVITY 2: HYPERVENTILATION
| back 4 THE pH VALUE BEGAN TO EXCEED THE NORMAL RANGE BETWEEN 10 AND 20 SECONDS - AS SOON AS IT ROSE ABOVE 7.45, THIS INDICATED THE CONDITION OF ALKALOSIS. |
front 5 ACTIVITY 2: HYPERVENTILATION
| back 5 IT WAS LARGER DURING HYPERVENTILATION |
front 6 ACTIVITY 2: HYPERVENTILATION
| back 6 THE TRACE FLAT-LINED, INDICATING THE BREATHING WAS SUSPENDED. BREATHING DID NOT RETURN TO NORMAL, IMMEDIATELY THE BODY TEMPORARILY STOPPED BREATHING IN ORDER TO RESTORE PCO2 LEVELS TO NORMAL VALUES. |
front 7 ACTIVITY 3: REBREATHING
| back 7 pH LEVELS OF BLOOD DECREASED OVER TIME WITH REBREATHING, AND pH LEVERS WEREN'T ALWAYS WITHIN NORMAL RANGE. |
front 8 ACTIVITY 3: REBREATHING
| back 8 REBREATHING RESULTED IN ACIDOSIS BECAUSE THE pH VALUE BEGAN TO DIP BELOW THE NORMAL RANGE BETWEEN 20 AND 30 SECONDS - SOON AS IT WENT BELOW 7.35. ACIDOSIS IS THE RESULT OF IMPAIRED RESPIRATION (HYPOVENTILATION) THAT LEADS TO THE ACCUMULATION OF TOO MUCH CARBON DIOXIDE IN THE BLOOD. |
front 9 ACTIVITY 3: REBREATHING
| back 9 LUNG DISEASE, AIRWAY OBSTRUCTION, DEPRESSING OF RESPIRATORY CENTER IN BRAIN STEM, AND DRUG OVERDOSE. |
front 10 ACTIVITY 4: RENAL RESPONSE TO NORMAL ACID-BASE BALANCE
| back 10 AS CO2 INCREASES, pH VALUE DECREASED. |
front 11 ACTIVITY 4: RENAL RESPONSE TO NORMAL ACID-BASE BALANCE
| back 11 BECAUSE CO2 LEVELS DIRECTLY AFFECT BLOOD pH LEVELS. |
front 12 ACTIVITY 5: RENAL RESPONSE TO RESPIRATORY ALKALOSIS
| back 12 A DECREASED LEVEL OF H+ WAS PRESENT IN THE URINE |
front 13 ACTIVITY 5: RENAL RESPONSE TO RESPIRATORY ALKALOSIS
| back 13 RENAL SYSTEM COMPENSATED FOR RESPIRATORY ALKALOSIS BY INCREASING PCO2 LEVELS AND DECREASING pH LEVELS. |
front 14 ACTIVITY 5: RENAL RESPONSE TO RESPIRATORY ALKALOSIS
| back 14 HYPERVENTILATION |
front 15 ACTIVITY 6: RENAL RESPONSE TO RESPIRATORY ACIDOSIS
| back 15 PENAL SYSTEM COMPENSATES FOR RESPIRATORY ACIDOSIS BY DECREASING PCO2 LEVELS AND INCREASING pH LEVELS. |
front 16 ACTIVITY 6: RENAL RESPONSE TO RESPIRATORY ACIDOSIS
| back 16 REBREATHING |
front 17 ACTIVITY 8: RESPIRATORY RESPONSE TO INCREASED METABOLISM
| back 17 METABOLIC WASTE PRODUCT |
front 18 ACTIVITY 8: RESPIRATORY RESPONSE TO INCREASED METABOLISM
| back 18 METABOLIC RATES OF 70 AND 80 |
front 19 ACTIVITY 8: RESPIRATORY RESPONSE TO INCREASED METABOLISM
| back 19 KETO ACIDOSIS, SALIVYLATE POISONING, INGESTING TOO MUCH ALCOHOL, DIARRHEA, AND STRENUOUS EXERCISE. |
front 20 ACTIVITY 9: RESPIRATORY RESPONSE TO DECREASED METABOLISM
| back 20 METABOLIC RATES OF 30 AND 20 |
front 21 ACTIVITY 9: RESPIRATORY RESPONSE TO DECREASED METABOLISM
| back 21 ALKALI INGESTING (ANTACIDS OR BICARBONATE), VOMITING, AND CONSTIPATION. |