front 1 hyposecretion of hormones from anterior pituitary gland; deficiency of one of the anterior pituitary hormones results in changes in metabolic or sexual function | back 1 hypopituitarism |
front 2 in children before the closure of the epiphyses, ______ (small stature) develops | back 2 dwarfism |
front 3 a lack of GH in adults does not affect bone length, but does affect bone | back 3 density and osteoporosis may develop |
front 4 clinical manifestations of decreased glucocorticoids associated with hypopituitarism? | back 4
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front 5 clinical manifestations of decreased mineralocorticoids associated with hypopituitarism? | back 5
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front 6 clinical manifestations of growth hormone associated with hypopituitarism? | back 6
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front 7 clinical manifestations of thyroid-stimulating hormone (TSH) associated with hypopituitarism? | back 7 think decreased levels of T3 and T4
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front 8 aldosterone controls? | back 8 Na+ and water |
front 9 lack of ACTH with a resultant decrease in glucocorticoids and mineralocorticoids is a life-threatening emergency because the pt is unable to maintain adequate fluid volume status, which may lead to circulatory collapse | back 9 panhypopituitarism |
front 10 what do pt's VS look like with hypopituitarism? | back 10
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front 11 what must we teach our patients about taking hormone at home? | back 11 taking hormone supplements in the morning mimics the normal release of these hormones |
front 12 hypersecretion of hormones from anterior pituitary gland; usually related to a hypersecreting tumor | back 12 hyperpituitarism |
front 13 in children before the closure of the epiphyses, _____ (large stature) develops | back 13 gigantism |
front 14 an excess of GH in adults does not affect bone length because of closure of the epiphyses but does affect bone density, and ______ (thickening of bones, particularly of the hands, feet, and facial bones) may develop | back 14 acromegaly |
front 15 clinical manifestations of increased glucocorticoids associated with hyperpituitarism? | back 15
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front 16 clinical manifestations of increased mineralocorticoids associated with hyperpituitarism? | back 16
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front 17 clinical manifestations of GH associated with hyperpituitarism? | back 17
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front 18 clinical manifestations of thyroid-stimulating hormone (TSH) associated with hyperpituitarism? | back 18 think increased T3 and T4
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front 19 vital signs with hyperpituitarism? | back 19
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front 20 review nursing interventions for a pt after transphenoidal hypophysectomy | back 20 pt 925 |
front 21 A nurse is caring for a client who is being evaluated for acromegaly. Which of the following manifestations should the nurse expect to find during assessment? Select all that apply. A. Loss of color discrimination B. Coarse facial features C. Enlarged distal extremities D. Hepatomegaly E. Moon face | back 21 A, B, C, D |
front 22 where are your adrenal glands located? | back 22 on top of each kidney |
front 23 the adrenal cortex secretes? | back 23
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front 24 primary glucocorticoid and its actions include carbohydrate, fat, and protein metabolism; suppression of the immune response; and control of the body's stress response | back 24 cortisol |
front 25 primary mineralocorticoid, and its primary actions are to promote sodium and water reabsorption and potassium excretion in the kidneys? | back 25 aldosterone |
front 26 what happens if exogenous corticosteroids are discontinued abruptly? | back 26 Addisonian crisis (acute adrenal crisis) |
front 27 adrenal insufficiency that may result from destruction of the adrenal glands or insufficiency or Addison's disease | back 27 adrenal cortical insufficiency |
front 28 adrenal cortical insufficiency (Addison's crisis) is a decrease/increase in glucocorticoids and mineralocorticoids | back 28 decrease |
front 29 patients presenting with acute adrenal insufficiency require emergency stabilization with IV fluids and glucose, along with IV administration of glucocorticoids | back 29 *** |
front 30 clinical manifestations of adrenal cortical insufficiency/Addison's crisis? | back 30
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front 31 what is our volume like in adrenal cortical insufficiency/Addison's crisis? | back 31 FVD
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front 32 what replacement is necessary for adrenal insufficiency? | back 32 replacement of cortisol |
front 33 The nurse correlates which clinical manifestation with the pathophysiology of adrenal insufficiency? A. Heat intolerance B. Weight gain C. Peripheral edema D. Hypoglycemia | back 33 D. Hypoglycemia |
front 34 A patient has been receiving doses of prednisone for treatment of RA for the past 3 months. If this medication is suddenly discontinued, for which complication is the patient at risk? A. Hypovolemia B. Hypernatremia C. Hypothermia D. Hyperglycemia | back 34 A. Hypovolemia |
front 35 adrenal cortex disorder in which there is an excessive secretion of glucocorticoids and mineralocorticoids | back 35 adrenal cortex hyperfunction; Cushing's |
front 36 clinical manifestations of adrenal cortex hyperfunction/Cushing's? | back 36
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front 37 clinical manifestations of adrenal cortex hyperfunction/Cushing's related to overproduction of cortisol? | back 37 decreased inflammatory and immune responses |
front 38 what is our fluid like in adrenal cortex hyperfunction/Cushing's? | back 38 FVE
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front 39 safety alert | back 39 pg 938 |
front 40 what are the electrolytes and BS doing in adrenal cortex hyperfunction/Cushing's? | back 40
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front 41 patients who have an adrenalectomy are at risk for? | back 41 Addison's; will need steroids |
front 42 possible complications from adrenal cortex hyperfunction/Cushing's? | back 42
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front 43 rare catecholamine secreting tumors of the adrenal medulla; secrete epi and norepi | back 43 pheochromocytoma |
front 44 clinical manifestations of pheochromocytoma? | back 44
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front 45 treatment for pheochromocytoma? | back 45 adrenalectomy is the definitive treatment |
front 46 vital signs in pheochromocytoma? | back 46
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front 47 pheochromocytoma is/is not life threatening | back 47 is life threatening |
front 48 what is important to remember about the morning of adrenalectomy? | back 48 administer glucocorticoid to avoid adrenal insufficiency |
front 49 A staff nurse is teaching a client who has Addison's disease about the disease process. The client asks the nurse what causes Addison's disease. Which of the following responses should the nurse make? A. It is caused by the lack of production of insulin by the pancreas. B. It is caused by the lack of production of aldosterone by the adrenal gland. C. It is caused by the overproduction of growth hormone by the pituitary gland. D. It is caused by the overproduction of parathormone by the parathyroid gland. | back 49 B. It is caused by the lack of production of aldosterone by the adrenal gland. |
front 50 A nurse is providing dietary teaching for a client who has Cushing's disease. Which of the following recommendations should the nurse include in the teaching? A. Limit intake of potassium-rich foods B. Restrict sodium intake C. Increase carbohydrate intake D. Decrease protein intake | back 50 B. Restrict sodium intake |
front 51 A nurse is caring for a client who has Cushing's syndrome. The nurse should recognize that which of the following are manifestations of Cushing's syndrome? (Select all that apply.) A. Alopecia B. Tremors C. Moon face D. Purple striations E. Buffalo hump | back 51 A, C, D, E |
front 52 A nurse is assessing a client who is admitted for elective surgery and has a history of Addison's disease. Which of the following findings should the nurse expect? A. Hyperpigmentation B. Intention tremors C. Hirsutism D. Purple striations | back 52 A. Hyperpigmentation |
front 53 A nurse is assessing four clients on a medical unit. The nurse should identify which of the following clients as exhibiting positive manifestations of hypercortiolism? A. A client who has a butterfly rash on his face B. Moon face C. A client who has a positive Chvostek's sign D. A client who has muscle hypertrophy | back 53 B. Moon face |